Phospho-p38 MAPK (Thr180/Tyr182) Antibody
Source: Rabbit pAb Catalog No. : CY6391
Phospho-p38 MAPK (Thr180/Tyr182) Antibody (CY6391)
UniProt:Q16539
Application:WB
Reactivity:Human,Mouse,Rat
Source:Rabbit pAb
References（4）
http://www.abways.cn/showproduct.asp?cid=CY6391
数据表
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UniProt
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Mol Weight43 kDa
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Alternative NamesCRK1, CSAID binding protein, CSBP, CSBP1, CSBP2, Cytokine suppressive anti- inflammatory drug binding protein, EC 2.7.11.24, MAP kinase MXI2, MAP kinase p38, MAPK14, MAX-interacting protein 2, MK14, MXI2,
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ImmunogenA synthesized peptide derived from human p38 MAPK around the phosphorylation site of Thr180/Tyr182) .
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StorageRabbit IgG in phosphate buffered saline , pH 7.4, 150mM NaCl, 0.02% sodium azide and 50% glycerol. Store at +4°C short term. Store at -20°C long term. Avoid freeze / thaw cycle.
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PurificationAffinity-chromatography
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IsotypeRabbit IgG
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ClonalityPolyclonal
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DescriptionThe protein encoded by this gene is a member of the MAP kinase family. MAP kinases act as an integration point for multiple biochemical signals, and are involved in a wide variety of cellular processes such as proliferation, differentiation, transcription regulation and development. This kinase is activated by various environmental stresses and proinflammatory cytokines.
Applications
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ReactivityHuman,Mouse,Rat
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ApplicationWB
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Recommended dilutionWB 1:500-1:2000 IHC 1:50-1:200 IF 1:200 IP
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Western blot analysis of p38 MAPK phosphorylation expression in Jurkat whole cell lysates.
引用文献（4条）
Euphorbia factor L2 alleviates lipopolysaccharide-induced acute lung injury and inflammation in mice through the suppression of NF-κB activation.-Biochemical Pharmacology
Exploring the Therapeutic Mechanism of Desmodium styracifolium on Oxalate Crystal-Induced Kidney Injuries Using Comprehensive Approaches Based on Proteomics and Network Pharmacology.-Front Pharmacol
Dl-3-n-butylphthalide exerts dopaminergic neuroprotection through inhibition of neuroinflammation.-Frontiers in Aging Neuroscience
LR12 Promotes Liver Repair by Improving the Resolution of Inflammation and Liver Regeneration in Mice with Thioacetamide- (TAA-) Induced Acute Liver Failure-Mediators of Inflammation (MEDIAT INFLAMM)
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